Herpes Zoster/Shingles:
Caused by reactivation of a latent varicella-zoster virus that resides in sensory ganglion – usually have history of previous varicella infection. More common in those who are immunocompromised.
Clinical features:
Pain and parathesias in dermatome followed by skin rash (vesiculopustular) – unilateral; vesicles are grouped on an erythematous base; associated with pain (eruption may be preceded by parathesia)
Treatment:
Acyclovir if immunocompromised.
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